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Dr. Heidi Noels and Dr. Yvonne Döring receive the W.H. Hauss Award from the DGAF

19-21 April 2018

Dr. Heidi Noels from the Institute for Molecular Cardiovascular Research at RWTH Aachen University together with Dr. Yvonne Döring from the Institute for Prophylaxis and Epidemiology of Circulatory Diseases at LMU Munich together received the DGAF W.H. Hauss Award 2018 from the German Society for Atherosclerosis Research (DGAF) during the 32nd Annual Meeting of the DGAF in Rauischholzhausen, Germany. They were awarded for their publication "Vascular CXCR4 Limits Atherosclerosis by Maintaining Arterial Integrity: Evidence From Mouse and Human Studies." (Circulation. 2017; 136: 388-403). In this work it could be shown that vascular CXCR4 has an atheroprotective function by maintaining the arterial integrity and preserving the endothelial barrier function. In addition, CXCR4 stabilizes a contractile smooth muscle cell phenotype. Targeted enhancement of these CXCR4-mediated protective functions could open up novel therapeutic options in the treatment of atherosclerosis.

The award was sponsored by a long-standing active member of the DGAF, Prof. Dr. Winfried März.

Robert Werner Mertens

MD student

University Hospital RWTH Aachen

Department of Internal Medicine

Project: The role of incretin hormone GLP-2 in septic cardiomyopathy

PI: Michael Lehrke

Robert Werner Mertens

MD student

University Hospital RWTH Aachen

Department of Internal Medicine

Project: The role of incretin hormone GLP-2 in septic cardiomyopathy

PI: Michael Lehrke

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Mechanisms of Cardiovascular Complications in Chronic Kidney Disease

PhD MD Students

David Schumacher

MD student

University Hospital RWTH Aachen

Institute for Molecular Cardiovascular Research

Project: The role of FGF23 after myocardial infarction

PI: Elisa Liehn

FGF23 is a protein mainly produced in osteocytes and acting on the kidney to regulate phosphate homeostasis and vitamin D metabolism. It has recently been shown to be directly correlated with cardiovascular mortality after myocardial infarction. However, the role of FGF23 in remodeling processes after acute myocardial infarction is still unknown.

We investigate the local role and function of FGF23 in a mouse model of myocardial infarction induced by left anterior descending artery ligation.

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